Neural signal through sympathetic component of autonomic nervous system (ANS) can

a. Increase the heart beat rate
b. Decrease the heart beat rate
c. Increase the strength of ventricular contraction
d. Decrease the speed of conduction of action potential
e. Increase cardiac output
f. Decrease cardiac output

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Solution

Effect of neural signals from ANS on heart

Under stress condition, the sympathetic nervous system directs adrenal glands to release norepinephrine hormone, also known as Emergency hormones or hormones of Fight or Flight.

Norepinephrine has the following effects on cardiac cells:

Increase in rate of heart beat

Increase in strength of ventricular contraction

Increase in cardiac output.

Final answer: (D) a, c and e

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Similar questions

The cardiac activity could be moderated by the autonomous neural system.
Tick the correct answer.
(a) The parasympathetic system stimulates heart rate and stroke volume
(b) The sympathetic system stimulates heart rate and stroke volume
(c) The parasympathetic system decrease the heart rate but increase stroke volume
(d) The sympathetic system decreases the heart rate but increases stroke volume

Q. Neural signals through parasympathetic neural signals (another component of ANS) can

a. Increase the heart beat rate
b. Decrease the heart beat rate
c. Increase the strength of ventricular contraction
d. Decrease the speed of conduction of action potentials
e. Increase the cardiac output
f. Decrease the cardiac output

Q. Assertion :Neural signals through sympathetic nerves can increases the strength of ventricular contraction. Reason: Parasympathetic neural signals synergistically act with sympathetic neural signal to increase the cardiac output.

Q. Statement 1: Parasympathetic neural system increases the rate of heart beat.
Statement 2: Sympathetic neural system decreases the rate of heart beat.

Q. Which of the following cardiac functions can be moderated by neural signals from special neural centre in the medulla oblongata?
a. Rate of heart beat
b. Cardiac output
c. Speed of conduction of action potential
d. Strength of ventricular contraction and thereby the cardiac output.

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